Tricaine methane-sulfonate (MS-222) application in fish anaesthesia

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Tricaine methane - sulfonate ( MS - 222 ) application in fish anaesthesia

Tricaine methane-sulfonate (MS-222) application in fish anaesthesia By N. Topic Popovic, I. Strunjak-Perovic, R. Coz-Rakovac, J. Barisic, M. Jadan, A. Persin Berakovic and R. Sauerborn Klobucar Laboratory of Ichthyopathology – Biological Materials, Division for Materials Chemistry, Rudjer Boskovic Institute, Zagreb, Croatia; Department of Anaesthesiology, University Hospital Clinic, Zagreb, Cro...

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Excitatory and inhibitory effects of tricaine (MS-222) on fictive breathing in isolated bullfrog brain stem.

This study examined the direct effects of tricaine methanesulfonate (MS-222), a sodium-channel blocking local anesthetic, on respiratory motor output using an in vitro brain stem preparation of adult North American bullfrogs (Rana catesbeiana). Bullfrogs were anesthetized with halothane, and the brain stem was removed and superfused with artificial cerebrospinal fluid containing MS-222 at conce...

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The Response of Gray Treefrogs to Anesthesia by Tricaine Methanesulfonate (TMS or MS-222)

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Behavioural Effects of the Commonly Used Fish Anaesthetic Tricaine Methanesulfonate (MS-222) on Zebrafish (Danio rerio) and Its Relevance for the Acetic Acid Pain Test

The pros and cons of using anaesthesia when handling fish in connection with experiments are debated. A widely adopted practice is to wait thirty minutes after anaesthesia before behavioural observations are initiated, but information about immediate effects of a treatment is then lost. This is pertinent for responses to acute stressors, such as acid injection in the acetic acid pain test. Howe...

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Efficacy of Tricaine Methanesulfonate (MS-222) as an Anesthetic Agent for Blocking Sensory-Motor Responses in Xenopus laevis Tadpoles

Anesthetics are drugs that reversibly relieve pain, decrease body movements and suppress neuronal activity. Most drugs only cover one of these effects; for instance, analgesics relieve pain but fail to block primary fiber responses to noxious stimuli. Alternately, paralytic drugs block synaptic transmission at neuromuscular junctions, thereby effectively paralyzing skeletal muscles. Thus, both ...

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ژورنال

عنوان ژورنال: Journal of Applied Ichthyology

سال: 2012

ISSN: 0175-8659

DOI: 10.1111/j.1439-0426.2012.01950.x